Escaping Anoikis through ROS: ANGPTL4 Controls Integrin Signaling through Nox1

Lance S. Terada; Fiemu E. Nwariaku

doi:10.1016/j.ccr.2011.02.019

Escaping Anoikis through ROS: ANGPTL4 Controls Integrin Signaling through Nox1

Lance S. Terada, Fiemu E. Nwariaku

Research output: Contribution to journal › Short survey › peer-review

18 Scopus citations

Abstract

Reactive oxygen species (ROS) mediate various cell fate decisions in normal and transformed cells. In this issue of Cancer Cell, Zhu et al. demonstrate the ability of ANGPTL4 to engage integrin-dependent survival signals by activation of the NADPH oxidase Nox1, thus mimicking anchorage conditions and bypassing anoikis by controlling ROS.

Original language	English (US)
Pages (from-to)	297-299
Number of pages	3
Journal	Cancer Cell
Volume	19
Issue number	3
DOIs	https://doi.org/10.1016/j.ccr.2011.02.019
State	Published - Mar 8 2011

ASJC Scopus subject areas

Oncology
Cell Biology
Cancer Research

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10.1016/j.ccr.2011.02.019

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title = "Escaping Anoikis through ROS: ANGPTL4 Controls Integrin Signaling through Nox1",

abstract = "Reactive oxygen species (ROS) mediate various cell fate decisions in normal and transformed cells. In this issue of Cancer Cell, Zhu et al. demonstrate the ability of ANGPTL4 to engage integrin-dependent survival signals by activation of the NADPH oxidase Nox1, thus mimicking anchorage conditions and bypassing anoikis by controlling ROS.",

author = "Terada, {Lance S.} and Nwariaku, {Fiemu E.}",

year = "2011",

month = mar,

day = "8",

doi = "10.1016/j.ccr.2011.02.019",

language = "English (US)",

volume = "19",

pages = "297--299",

journal = "Cancer Cell",

issn = "1535-6108",

publisher = "Cell Press",

number = "3",

}

TY - JOUR

T1 - Escaping Anoikis through ROS

T2 - ANGPTL4 Controls Integrin Signaling through Nox1

AU - Terada, Lance S.

AU - Nwariaku, Fiemu E.

PY - 2011/3/8

Y1 - 2011/3/8

N2 - Reactive oxygen species (ROS) mediate various cell fate decisions in normal and transformed cells. In this issue of Cancer Cell, Zhu et al. demonstrate the ability of ANGPTL4 to engage integrin-dependent survival signals by activation of the NADPH oxidase Nox1, thus mimicking anchorage conditions and bypassing anoikis by controlling ROS.

AB - Reactive oxygen species (ROS) mediate various cell fate decisions in normal and transformed cells. In this issue of Cancer Cell, Zhu et al. demonstrate the ability of ANGPTL4 to engage integrin-dependent survival signals by activation of the NADPH oxidase Nox1, thus mimicking anchorage conditions and bypassing anoikis by controlling ROS.

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U2 - 10.1016/j.ccr.2011.02.019

DO - 10.1016/j.ccr.2011.02.019

M3 - Short survey

C2 - 21397852

AN - SCOPUS:79952524783

SN - 1535-6108

VL - 19

SP - 297

EP - 299

JO - Cancer Cell

JF - Cancer Cell

IS - 3

ER -

Escaping Anoikis through ROS: ANGPTL4 Controls Integrin Signaling through Nox1

Abstract

ASJC Scopus subject areas

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