Escaping Anoikis through ROS

ANGPTL4 Controls Integrin Signaling through Nox1

Research output: Contribution to journalArticle

14 Citations (Scopus)

Abstract

Reactive oxygen species (ROS) mediate various cell fate decisions in normal and transformed cells. In this issue of Cancer Cell, Zhu et al. demonstrate the ability of ANGPTL4 to engage integrin-dependent survival signals by activation of the NADPH oxidase Nox1, thus mimicking anchorage conditions and bypassing anoikis by controlling ROS.

Original languageEnglish (US)
Pages (from-to)297-299
Number of pages3
JournalCancer Cell
Volume19
Issue number3
DOIs
StatePublished - Mar 8 2011

Fingerprint

Anoikis
Integrins
Reactive Oxygen Species
NADPH Oxidase
Neoplasms

ASJC Scopus subject areas

  • Cancer Research
  • Cell Biology
  • Oncology

Cite this

Escaping Anoikis through ROS : ANGPTL4 Controls Integrin Signaling through Nox1. / Terada, Lance S.; Nwariaku, Fiemu E.

In: Cancer Cell, Vol. 19, No. 3, 08.03.2011, p. 297-299.

Research output: Contribution to journalArticle

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