Progression of Diabetic Retinopathy after Pancreas Transplantation

Philip Raskin; J. Rosenstock; R. M. Cohen; M. A. Berk; Z. Rymaszewski; M. J. Lipman; J. Benn; A. Konigsrainer; K. Miller; G. Kieselbach; R. Margreiter; R. C. Ramsay; F. C. Goetz; D. E R Sutherland

doi:10.1056/NEJM198807283190414

Progression of Diabetic Retinopathy after Pancreas Transplantation

Philip Raskin, J. Rosenstock, R. M. Cohen, M. A. Berk, Z. Rymaszewski, M. J. Lipman, J. Benn, A. Konigsrainer, K. Miller, G. Kieselbach, R. Margreiter, R. C. Ramsay, F. C. Goetz, D. E R Sutherland

Research output: Contribution to journal › Letter › peer-review

6 Scopus citations

Abstract

To the Editor: We were not surprised to read in the article by Ramsay et al. (Jan. 28 issue)¹ that restoration of metabolic control by pancreas transplantation failed to halt the progression of diabetic retinopathy. Most of the patients, to judge from the description given in the paper, must have had advanced and theoretically irreversible diabetic retinopathy. Furthermore, it is conceivable that even if hyperglycemia is necessary for the development of diabetic microvascular complications, individual genetic susceptibility in response to the metabolic derangements varies considerably.² We would like to point out one misquotation in the discussion of the paper. The.

Original language	English (US)
Pages (from-to)	247-249
Number of pages	3
Journal	New England Journal of Medicine
Volume	319
Issue number	4
DOIs	https://doi.org/10.1056/NEJM198807283190414
State	Published - Jul 28 1988

ASJC Scopus subject areas

General Medicine

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Raskin, P., Rosenstock, J., Cohen, R. M., Berk, M. A., Rymaszewski, Z., Lipman, M. J., Benn, J., Konigsrainer, A., Miller, K., Kieselbach, G., Margreiter, R., Ramsay, R. C., Goetz, F. C., & Sutherland, D. E. R. (1988). Progression of Diabetic Retinopathy after Pancreas Transplantation. New England Journal of Medicine, 319(4), 247-249. https://doi.org/10.1056/NEJM198807283190414

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abstract = "To the Editor: We were not surprised to read in the article by Ramsay et al. (Jan. 28 issue)1 that restoration of metabolic control by pancreas transplantation failed to halt the progression of diabetic retinopathy. Most of the patients, to judge from the description given in the paper, must have had advanced and theoretically irreversible diabetic retinopathy. Furthermore, it is conceivable that even if hyperglycemia is necessary for the development of diabetic microvascular complications, individual genetic susceptibility in response to the metabolic derangements varies considerably.2 We would like to point out one misquotation in the discussion of the paper. The.",

author = "Philip Raskin and J. Rosenstock and Cohen, {R. M.} and Berk, {M. A.} and Z. Rymaszewski and Lipman, {M. J.} and J. Benn and A. Konigsrainer and K. Miller and G. Kieselbach and R. Margreiter and Ramsay, {R. C.} and Goetz, {F. C.} and Sutherland, {D. E R}",

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T1 - Progression of Diabetic Retinopathy after Pancreas Transplantation

AU - Raskin, Philip

AU - Rosenstock, J.

AU - Cohen, R. M.

AU - Berk, M. A.

AU - Rymaszewski, Z.

AU - Lipman, M. J.

AU - Benn, J.

AU - Konigsrainer, A.

AU - Miller, K.

AU - Kieselbach, G.

AU - Margreiter, R.

AU - Ramsay, R. C.

AU - Goetz, F. C.

AU - Sutherland, D. E R

PY - 1988/7/28

Y1 - 1988/7/28

N2 - To the Editor: We were not surprised to read in the article by Ramsay et al. (Jan. 28 issue)1 that restoration of metabolic control by pancreas transplantation failed to halt the progression of diabetic retinopathy. Most of the patients, to judge from the description given in the paper, must have had advanced and theoretically irreversible diabetic retinopathy. Furthermore, it is conceivable that even if hyperglycemia is necessary for the development of diabetic microvascular complications, individual genetic susceptibility in response to the metabolic derangements varies considerably.2 We would like to point out one misquotation in the discussion of the paper. The.

AB - To the Editor: We were not surprised to read in the article by Ramsay et al. (Jan. 28 issue)1 that restoration of metabolic control by pancreas transplantation failed to halt the progression of diabetic retinopathy. Most of the patients, to judge from the description given in the paper, must have had advanced and theoretically irreversible diabetic retinopathy. Furthermore, it is conceivable that even if hyperglycemia is necessary for the development of diabetic microvascular complications, individual genetic susceptibility in response to the metabolic derangements varies considerably.2 We would like to point out one misquotation in the discussion of the paper. The.

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Progression of Diabetic Retinopathy after Pancreas Transplantation

Abstract

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