Regulation of PERK-eIF2α signalling by tuberous sclerosis complex-1 controls homoeostasis and survival of myelinating oligodendrocytes

Physics & Astronomy

• homeostasis 81%
• endoplasmic reticulum 79%
• myelin 42%
• phosphatases 41%
• deletion 38%
• abnormalities 36%
• mutations 32%
• death 31%
• defects 30%
• mice 29%
• ablation 26%
• activation 22%
• causes 16%
• augmentation 16%

Medicine & Life Sciences

• Tuberous Sclerosis 1 96%
• Tuberous Sclerosis 69%
• Oligodendroglia 61%
• Homeostasis 40%
• Tuberous Sclerosis 2 37%
• Endoplasmic Reticulum Stress 24%
• Guanabenz 18%
• Oligodendrocyte Precursor Cells 14%
• MAP Kinase Signaling System 12%
• Myelin Sheath 11%
• Phosphoric Monoester Hydrolases 11%
• White Matter 10%
• Cell Death 8%
• Mutation 6%

Chemical Compounds

• Guanabenz 100%
• Ablation 64%
• Apoptotic 64%
• Cell Death 59%
• Mutation 47%
• White 46%