The cellular basis of Fanconi syndrome

Research output: Contribution to journalReview article

13 Scopus citations

Abstract

In this syndrome, all solutes are prevented from crossing the apical membrane of the proximal renal tubule cell; hence, investigations have focused on generalized transport dysfunction, namely intracellular ATP activity. Experiments suggest that intracellular adenine nucleotide degradation secondary to phosphate depletion causes a decline in ATP.

Original languageEnglish (US)
Pages (from-to)137-140+142+147-148
JournalHospital Practice
Volume28
Issue number11
DOIs
StatePublished - 1993

ASJC Scopus subject areas

  • Medicine(all)

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