Inflammation-induced caveolin-1 and BMPRII depletion promotes endothelial dysfunction and TGF-β-driven pulmonary vascular remodeling

Medicine & Life Sciences

• Type II Bone Morphogenetic Protein Receptors 100%
• Endothelial Growth Factors 90%
• Caveolin 1 80%
• Vascular Remodeling 73%
• Transforming Growth Factors 65%
• Endothelial Cells 40%
• Lipopolysaccharides 40%
• Inflammation 36%
• Lung 33%
• Adult Respiratory Distress Syndrome 17%
• Acute Lung Injury 8%
• Nitric Oxide Synthase Type III 8%
• Vascular System Injuries 8%
• Serum Albumin 7%
• Interleukin-6 5%
• Oxidative Stress 5%
• Homeostasis 5%
• Escherichia coli 5%
• Phosphorylation 5%
• Blood Vessels 4%