Neuroimaging, cognitive, and neurobehavioral outcomes following carbon monoxide poisoning

Ramona O. Hopkins, Fu Lye M Woon

Research output: Contribution to journalArticle

37 Citations (Scopus)

Abstract

Carbon monoxide is a colorless, odorless gas produced as a byproduct of combustion. Carbon monoxide is the leading cause of poisoning injury and death worldwide. Morbidity following CO poisoning includes neurologic sequelae, neuropathologic abnormalities on brain imaging, neurobehavioral changes, and cognitive impairments. It is estimated that as high as 50% of individuals with carbon monoxide poisoning will develop neurologic, neurobehavioral, or cognitive sequelae. Carbon monoxide related cognitive impairments included impaired memory, attention, executive function, motor, visual spatial, and slow mental processing speed. Given the high rate of brain related morbidity and the fact that the majority of carbon monoxide is avoidable, awareness and prevention of carbon monoxide poisoning is warranted.

Original languageEnglish (US)
Pages (from-to)141-155
Number of pages15
JournalBehavioral and Cognitive Neuroscience Reviews
Volume5
Issue number3
DOIs
StatePublished - Sep 2006

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Carbon Monoxide Poisoning
Carbon Monoxide
Neuroimaging
Poisoning
Nervous System
Morbidity
Executive Function
Gases
Wounds and Injuries
Brain

Keywords

  • Anxiety
  • Brain imaging
  • Carbon monoxide poisoning
  • Cognitive impairments
  • Depression

ASJC Scopus subject areas

  • Behavioral Neuroscience
  • Cognitive Neuroscience

Cite this

Neuroimaging, cognitive, and neurobehavioral outcomes following carbon monoxide poisoning. / Hopkins, Ramona O.; Woon, Fu Lye M.

In: Behavioral and Cognitive Neuroscience Reviews, Vol. 5, No. 3, 09.2006, p. 141-155.

Research output: Contribution to journalArticle

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