Omega 3 fatty acids and GPR120

Dayoung Oh; Jerrold M. Olefsky

doi:10.1016/j.cmet.2012.04.009

Omega 3 fatty acids and GPR120

Dayoung Oh, Jerrold M. Olefsky

Research output: Contribution to journal › Short survey › peer-review

77 Scopus citations

Abstract

Human loss-of-function gene variants in GPR120 have recently been identified that confer increased risk for obesity and metabolic syndrome. In addition, GPR120 KO mice develop obesity, increased inflammation, and insulin resistance, consistent with a role for GPR120 signaling in the metabolic syndrome and diabetes mellitus.

Original language	English (US)
Pages (from-to)	564-565
Number of pages	2
Journal	Cell Metabolism
Volume	15
Issue number	5
DOIs	https://doi.org/10.1016/j.cmet.2012.04.009
State	Published - May 2 2012

ASJC Scopus subject areas

Physiology
Molecular Biology
Cell Biology

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10.1016/j.cmet.2012.04.009

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title = "Omega 3 fatty acids and GPR120",

abstract = "Human loss-of-function gene variants in GPR120 have recently been identified that confer increased risk for obesity and metabolic syndrome. In addition, GPR120 KO mice develop obesity, increased inflammation, and insulin resistance, consistent with a role for GPR120 signaling in the metabolic syndrome and diabetes mellitus.",

author = "Dayoung Oh and Olefsky, {Jerrold M.}",

year = "2012",

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doi = "10.1016/j.cmet.2012.04.009",

language = "English (US)",

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journal = "Cell Metabolism",

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T1 - Omega 3 fatty acids and GPR120

AU - Oh, Dayoung

AU - Olefsky, Jerrold M.

PY - 2012/5/2

Y1 - 2012/5/2

N2 - Human loss-of-function gene variants in GPR120 have recently been identified that confer increased risk for obesity and metabolic syndrome. In addition, GPR120 KO mice develop obesity, increased inflammation, and insulin resistance, consistent with a role for GPR120 signaling in the metabolic syndrome and diabetes mellitus.

AB - Human loss-of-function gene variants in GPR120 have recently been identified that confer increased risk for obesity and metabolic syndrome. In addition, GPR120 KO mice develop obesity, increased inflammation, and insulin resistance, consistent with a role for GPR120 signaling in the metabolic syndrome and diabetes mellitus.

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U2 - 10.1016/j.cmet.2012.04.009

DO - 10.1016/j.cmet.2012.04.009

M3 - Short survey

C2 - 22560206

AN - SCOPUS:84860476011

SN - 1550-4131

VL - 15

SP - 564

EP - 565

JO - Cell Metabolism

JF - Cell Metabolism

IS - 5

ER -

Omega 3 fatty acids and GPR120

Abstract

ASJC Scopus subject areas

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