Omega 3 fatty acids and GPR120

Dayoung Oh, Jerrold M. Olefsky

Research output: Contribution to journalArticle

56 Scopus citations

Abstract

Human loss-of-function gene variants in GPR120 have recently been identified that confer increased risk for obesity and metabolic syndrome. In addition, GPR120 KO mice develop obesity, increased inflammation, and insulin resistance, consistent with a role for GPR120 signaling in the metabolic syndrome and diabetes mellitus.

Original languageEnglish (US)
Pages (from-to)564-565
Number of pages2
JournalCell Metabolism
Volume15
Issue number5
DOIs
StatePublished - May 2 2012

    Fingerprint

ASJC Scopus subject areas

  • Cell Biology
  • Molecular Biology
  • Physiology

Cite this